首页良性前列腺增生病因/危险因素证据详情

Lower thiol, glutathione, and glutathione peroxidase levels in prostate cancer: a meta-analysis study

原文:2020年 发布于 Aging Male 23卷 第5期 1533-1544 浏览量:1022次 原文链接

作者: Sajjaboontawee N. Supasitthumrong T. Tunvirachaisakul C. Nantachai K. Snabboon T. Reiche E. M. V. Simão A. N. C. Maes M.

作者单位: Faculty of Medicine, Department of Psychiatry, Chulalongkorn University, Bangkok, Thailand. Faculty of Medicine, Department of Medicine, Chulalongkorn University, Bangkok, Thailand. Department of Pathology, Clinical Analysis and Toxicology, Health Sciences Center, Londrina State University, Londrina, Brazil. Department of Psychiatry, Medical University of Plovdiv, Plovdiv, Bulgaria. IMPACT Strategic Research Centre, Deakin University, Geelong, Australia.

归属分类: 良性前列腺增生病因/危险因素证据

DOI: 10.1080/13685538.2020.1858048

关键词: Glutathione Glutathione Peroxidase Humans Male *Prostatic Hyperplasia *Prostatic Neoplasms Sulfhydryl Compounds Antioxidants benign prostatic hyperplasia biomarkers oxidative stress prostate cancer

文献简介

PURPOSE: Lowered thiol (-SH) groups and glutathione (GSH) metabolism may be associated with prostate cancer (PCa) and benign prostatic hyperplasia (BPH). The objectives of this study were to systematically review and meta-analyze the associations among -SH groups, GSH, GSH peroxidase (GPx), GSH reductase (GR), and GSH transferase (GST) and PCa/BPH. METHODS: Four electronic databases were searched for studies that reported -SH and GSH variables in PCa/BPH and healthy controls (HC) and the data were meta-analyzed by calculating Hedges's g with 95% confidence intervals. RESULTS: Twenty studies were included in this meta-analysis. Total -SH (g = -1.750, -2.341/-1.159), GPx (g = -0.789, -1.234/-0.344), GSH (g = -2.219, -4.132/-0.305), and the combination of -SH, GPx, and GSH (g = -1.271, -1.271/-0.800) were significantly lower in PCa patients than in HC. -SH (g = -1.752, -3.123/-0.381) and the combination of -SH, GPx, and GSH (g = -0.813, -1.298/-0.327) were significantly lower in BPH patients than in HC. GPx was significantly lower in PCa than in BPH patients (g = -0.455, -0.896/-0.014). Heterogeneity levels were very high, but Egger's test showed that none of the biomarkers showed significant publication bias. CONCLUSION: Thiol/GPx antioxidant defenses are significantly attenuated in patients with PCa while patients with BPH occupy an intermediate risk group position between PCa patients and HC.

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