首页膀胱肿瘤诊断证据详情

The relationship between GSTA1, GSTM1, GSTP1, and GSTT1 genetic polymorphisms and bladder cancer susceptibility: A meta-analysis

原文: 2016 年 发布于 Bladder Cancer 浏览量:168次

作者: Yu Y. J. Li X. Liang C. Tang J. Y. Qin Z. Q. Wang C. M. Xu W. Z. Hua Y. B. Shao P. F. Xu T.

作者单位: Department of Urology, Hospital Universitario Fundación de Alcorcón, Madrid, Spain. Electronic address: vhernandez@fhalcorcon.es. Department of Urology, Hospital Universitario Infanta Sofía, Madrid, Spain. Department of Urology, Derriford Hospital, Plymouth UK. Academic Urology Unit, University of Aberdeen, Scotland, UK. Department of Medicine, Health Science Centre, McMaster University, Hamilton, ON, Canada. Department of Pathology, Groupe Hospitalier Pitié-Salpêtrière, Paris, France. Department of Radiology, Queen Alexandra Hospital, Portsmouth, UK. Department of Urology, Eberhard-Karls University, Tübingen, Germany. Department of Urology, Foch Hospital, Suresnes, France. Department of Urology, Radboud University Medical Center, Nijmegen, The Netherlands. Department of Urology, Hospital Clinic, University of Barcelona, Barcelona, Spain.

归属分类: 膀胱肿瘤诊断证据

DOI: 10.1186/s12885-018-5014-1

文献简介

CONTEXT: Tobacco smoke includes a mix of carcinogens implicated in the etiology of bladder cancer (BC) and renal cell cancer (RCC). OBJECTIVE: We reviewed the impact of tobacco exposure on BCC and RCC incidence and mortality, and whether smoking cessation decreases the risk. EVIDENCE ACQUISITION: A systematic review of original articles in English was performed in August 2013. Meta-analysis of risks was performed using adjusted risk ratios where available. Publication bias was assessed using Begg and Egger tests. EVIDENCE SYNTHESIS: We identified 2683 papers, of which 107 fulfilled our inclusion criteria, of which 83 studies investigated BC and 24 investigated RCC. The pooled relative risk (RR) of BC incidence was 2.58 (95% confidence interval [CI] 2.37-2.80) for all smokers, 3.47 (3.07-3.91) for current smokers, and 2.04 (1.85-2.25) for former smokers. The corresponding pooled RR of BC disease-specific mortality (DSM) was 1.47 (1.24-1.75), 1.53 (1.12-2.09) and 1.44 (0.99-2.11). The pooled RR of RCC incidence was 1.31 (1.22-1.40) for all smokers, 1.36 (1.19-1.56) for current smokers, and 1.16 (1.08-1.25) for former smokers. The corresponding RCC DSM risk was 1.23 (1.08-1.40), 1.37 (1.19-1.59), and 1.02 (0.90-1.15). CONCLUSIONS: We present an up-to-date review of tobacco smoking and BC and RCC incidence and mortality. Tobacco smoking significantly increases the risk of BC and RCC incidence. BC incidence and DSM risk are greatest in current smokers and lowest in former smokers, indicating that smoking cessation confers benefit. We found that secondhand smoke exposure is associated with a significant increase in BC risk. PATIENT SUMMARY: Tobacco smoking affects the development and progression of bladder cancer and renal cell cancer. Smoking cessation reduces the risks of developing and dying from these common cancers. We quantify these risks using the most up-to-date results published in the literature.

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